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Metco (Metronidazole)
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Metco

Metco eliminates bacteria and other microorganisms that cause infections of the reproductive system, gastrointestinal tract, skin, vagina, and other areas of the body. Antibiotics will not work for colds, flu, or other viral infections. This medication is sometimes prescribed for other uses; ask your doctor or pharmacist for more information.

Other names for this medication:
Aldezole, Anabact, Anaeromet, Anerobia, Aristogyl, Avidal, Biatron, Clont, Corsagyl, Cresac, Dequazol, Dirozyl, Efloran, Farnat, Flagystatin, Flazol, Flegyl, Geloderm, Medamet, Medazol, Metrajil, Metrocream, Metrodal, Metronex, Metronidazole, Metronidazols, Metroson, Micogyl, Negazole, Nidazol, Nipazol, Patryl, Perilox, Polibiotic, Protogyl, Rosasol, Rosazol, Rozagel, Rupezol, Suanatem, Trichodazol, Tricodazol, Trikozol, Vertisal, Wingyl

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Also known as: Metronidazole

Description

Generic Metco is created by pharmacy specialists to struggle with dangerous infections spread by bacteria (it can be protozoa or anaerobic bacteria). Target of Generic Metco is to control, ward off and terminate bacteria. Generic Metco acts as an anti-infection remedy. Generic Metco operates by killing bacteria which spreads by infection. Metco is also known as Metronidazole. Generic Metco and other antibiotics don"t treat viral infections (flu, cold and other). Generic Metco also does not help with vaginal yeast infection. Generic name of Generic Metco is Metronidazole. Brand names of Generic Metco are Protostat, Metco, Metco ER, Metco 375.

Dosage

Metco 250-mg tablets are round, blue, film coated, with SEARLE and 1831 debossed on one side and Metco and 250 on the other side; bottles of 50, 100, and 2,500. Metco 500-mg tablets are oblong, blue, film coated, with Metco debossed on one side and 500 on the other side; bottles of 50, 100, and 500.

Overdose

Single oral doses of Metco, up to 15 g, have been reported in suicide attempts and accidental overdoses. Symptoms reported include nausea, vomiting, and ataxia. Oral Metco has been studied as a radiation sensitizer in the treatment of malignant tumors. Neurotoxic effects, including seizures and peripheral neuropathy, have been reported after 5 to 7 days of doses of 6 to 10.4 g every other day.

There is no specific antidote for Metco overdose; therefore, management of the patient should consist of symptomatic and supportive therapy.

Storage

Store at room temperature below 25 degrees C (77 degrees F) away from moisture, light and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Metco are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Metronidazole is mainly metabolised by hepatic oxidation. Substantial impairment of Metronidazole clearance may occur in the presence of advanced hepatic insufficiency. The risk/benefit ratio of using Metronidazole to treat trichomoniasis in such patients should be carefully considered. Plasma levels of Metronidazole should be closely monitored.

Cases of severe hepatotoxicity/acute hepatic failure, including cases with a fatal outcome with very rapid onset after treatment initiation in patients with Cockayne syndrome have been reported with products containing metronidazole for systemic use. In this population, metronidazole should therefore be used after careful benefit-risk assessment and only if no alternative treatment is available. Liver function tests must be performed just prior to the start of therapy, throughout and after end of treatment until liver function is within normal ranges, or until the baseline values are reached. If the liver function tests become markedly elevated during treatment, the drug should be discontinued.

Patients with Cockayne syndrome should be advised to immediately report any symptoms of potential liver injury to their physician and stop taking metronidazole.

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Characterization of TCHQ-induced genotoxicity and mutagenesis using the pSP189 shuttle vector in mammalian cells. These phenotypes of the mutant varied substantially with light conditions. The degree of chromosome damage was directly proportional to the concentrations used for zinc sulfate and zinc acetate but not for zinc chloride. Standard hospital reporting procedures were proposed as a means of improving identification. The overall mortality is 53 per cent and is higher in patients with early versus late PVE. Complications that include visual acuity decline, prolonged anterior chamber inflammation, phthisis bulbi, scleral thinning and ocular pain were noted. aeruginosa antimicrobial drug resistance at six French hospitals over a longer period of time (2001-2011) and with a constant definition of resistance. Analyses of the distinct calculations show that the conformation of cytochrome f is strained upon binding of its partner, and relaxes upon its release. Linear and logarithmic coherent spatial-filtering techniques are compared in the separation of multiplicative noise from an image in a coherent-optical image-processing system. An HIV positive patient with extensive destructions in the midline area of maxilla and mandible is presented.

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The subgroup of patients with head tremor experienced the most gait and balance difficulty, with nearly one-in-two having had multiple near misses or falls during the previous year. Circulating PD-L1/PD-1 expression was tested by flow cytometry, and 23 patients were simultaneously evaluated for intratumoral PD-L1 expression by immunohistochemical staining. Analysis of CML fluxes showed no release/uptake of CML by the liver. To evaluate effects of postmenopausal hormone replacement therapy (HRT) on von Willebrand factor, factor (F)VIII, factor (F)VII, fibrinogen, antithrombin (AT) III, prothrombin fragments 1 and 2, protein C, total and free protein S, plasminogen activator inhibitor-1 (PAI-1), tissue plasminogen activator (tPA) and resistance to activated protein C. More importantly, dsP53-285 suppressed PCa cells mainly by modulating wild-type p53 expression. At necropsy, the experimental animals and the normal dog showed viable chondrocytes and osteocytes, with minimal bone necrosis. Degloving injuries may be a challenge when it comes to deciding the surgical approach to be used.

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